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ex vivo langendorff isolated heart perfusion system  (Radnoti LLC)

 
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  • 90

    Structured Review

    Radnoti LLC ex vivo langendorff isolated heart perfusion system
    Ex Vivo Langendorff Isolated Heart Perfusion System, supplied by Radnoti LLC, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/ex+vivo+langendorff+isolated+heart+perfusion+system/ex+vivo+langendorff+isolated+heart+perfusion+system/pm36549501-67-7-14
    Average 90 stars, based on 1 article reviews
    ex vivo langendorff isolated heart perfusion system - by Bioz Stars, 2026-09
    90/100 stars

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    Related Articles

    Ex Vivo:

    Article Title: Succinate level is increased and succinate dehydrogenase exerts forward and reverse catalytic activities in lipopolysaccharides-stimulated cardiac tissue: The protective role of dimethyl malonate.
    Article Snippet: This study aimed to investigate the alterations of myocardial succinate and fumarate levels with or without succinate dehydrogenase (SDH) inhibitor dimethyl malonate during 24 h of lipopolysaccharides (LPS) challenge, as well as the effects of dimethyl malonate on the impaired cardiac tissue.. Myocardial succinate and fumarate levels were increased in the initial 9 h of LPS challenge.. During this time, dimethyl malonate increased the succinate level, decreased the fumarate level, aggravated the cardiac dysfunction, reduced the oxidative stress, had little effect on interleukin-1β production, promoted interleukin-10 production and bothered the ATP production.

    Isolation:

    Article Title: Succinate level is increased and succinate dehydrogenase exerts forward and reverse catalytic activities in lipopolysaccharides-stimulated cardiac tissue: The protective role of dimethyl malonate.
    Article Snippet: This study aimed to investigate the alterations of myocardial succinate and fumarate levels with or without succinate dehydrogenase (SDH) inhibitor dimethyl malonate during 24 h of lipopolysaccharides (LPS) challenge, as well as the effects of dimethyl malonate on the impaired cardiac tissue.. Myocardial succinate and fumarate levels were increased in the initial 9 h of LPS challenge.. During this time, dimethyl malonate increased the succinate level, decreased the fumarate level, aggravated the cardiac dysfunction, reduced the oxidative stress, had little effect on interleukin-1β production, promoted interleukin-10 production and bothered the ATP production.



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